Special Forces- Science Explains Their Reaction To Stress

This is a very interesting article about why some people handle stress better than others do. Dr. Andy Morgan of Yale Medical School conducted research to determine why some people handle stress better than others do.

His research took him to Fort Bragg which where the Army's elite Airborne and Special Forces school is located. Let's take a look at an excerpt from the article.

"For Morgan, POW school was the perfect place to study who survives the best under acute stress. If you think it's just training and the soldiers know they're not really in serious danger, consider what Morgan discovered. During mock interrogations, the prisoners' heart rates skyrocket to more than 170 beats per minute for more than half an hour, even though they aren't engaging in any physical activity. Meanwhile, their bodies pump more stress hormones than the amounts actually measured in aviators landing on aircraft carriers, troops awaiting ambushes in Vietnam, skydivers taking the plunge or patients awaiting major surgery. The levels of stress hormones are sufficient to turn off the immune system and to produce a catabolic state, in which the body begins to break down and feed on itself. The average weight loss in three days is 22 pounds.

Morgan's research—the first of its kind—produced some fascinating findings about who does the best job resisting the interrogators and who stays focused and clearheaded despite the uncontrollable fear. Morgan looked at two different groups going through this training: regular Army troops like infantrymen, and elite Special Forces soldiers, who are known to be especially "stress hardy" or cool under pressure. At the start or base line, the two groups were essentially the same, but once the stress began, and afterward, there were significant differences. Specifically, the two groups released very different amounts of a chemical in the brain called neuropeptide Y. NPY is an abundant amino acid in our bodies that helps regulate our blood pressure, appetite, learning and memory. It also works as a natural tranquilizer, controlling anxiety and buffering the effects of stress hormones like norepenephrine, one of the chemicals that most of us simply call adrenaline. In essence, NPY is one of the fire hoses that your brain uses to extinguish your alarm and fear responses by keeping the frontal-lobe parts of your brain working longer under stress.

Morgan found one very specific reason that Special Forces are superior survivors: they produce significantly greater levels of NPY compared with regular troops. In addition, 24 hours after completing survival training, Special Forces soldiers returned to their original levels of NPY while regular soldiers were significantly below normal."

So if this NPY serves as a sort of natural anti-anxiety drug I have to ask the obvious question. Can we find a way to produce it? Maybe I am being naive, but from a laymen's perspective it sounds like it could be a great resource for people. It might allow some people to stop taking their meds.

Another part of the article that I thought was interesting is the section in which they discussed heart rate variability. Take a look at this:

It turns out that the best survivors don't have a lot of heart-rate variability. Instead, they've got "metronomic heartbeats"—their hearts thump steadily like metronomes—with almost no variability between beats. That is, the intervals between the beats are evenly spaced. Morgan believes that a metronomic heartbeat is an easy way to detect good survivors and high neuropeptide Y releasers. It makes sense biologically because your brainstem, which controls your heartbeat, has a high density of neuropeptide Y.

Part of what I found interesting was that the article says that metronomic heartbeat is associated with early heart disease and sudden death. So there is a question about whether this is really a benefit. It is good if you are a soldier or in some sort of very stressful profession.

But if it is tied to heart disease the negative can potentially outweigh the positive. Nice to stay calm, but not at the expense of not living past fifty.

Potential Health Benefits of Blueberries

I thought that this article on WebMD was interesting.

In the study, presented at Experimental Biology 2009, researchers fed rats bred to become obese either a high-fat or low-fat diet enriched with whole blueberry powder or carbohydrates as 2% of their total diet.

After 90 days, the rats fed blueberries had less abdominal fat, lower cholesterol, and improved glucose control and insulin sensitivity. The latter two factors are markers of how well the body processes sugar for energy and are related to diabetes risk.

These health benefits of blueberries were evident in rats fed both high- and low-fat diets enriched with the blueberry powder. But the benefits were greatest among those who ate a low-fat diet.

In addition to the other heart health benefits of blueberries, those fed the low-fat blueberry diet also lost body weight and fat mass compared to those on the high-fat diet.

Although more research is needed to confirm these results in humans, a related study presented at the same conference showed that men with risk factors for heart disease who drank wild blueberry juice for three weeks seemed to experience slight improvements in glucose and insulin control.

Should You Be Scared about The Swine Flu

Take a look at this CNN story:

(CNN) -- There had been no confirmed deaths in the United States related to swine flu as of Tuesday afternoon. But another virus had killed thousands of people since January and is expected to keep killing hundreds of people every week for the rest of the year.

That one? The regular flu.

An outbreak of swine flu that is suspected in more than 150 deaths in Mexico and has sickened dozens of people in the United States and elsewhere has grabbed the attention of a nervous public and of medical officials worried the strain will continue to mutate and spread.
Experts are nervous that, as a new strain, the swine flu will be harder to stop because there aren't any vaccines to fight it.

But even if there are swine-flu deaths outside Mexico -- and medical experts say there very well may be -- the virus would have a long way to go to match the roughly 36,000 deaths that seasonal influenza causes in the United States each year.

"That happens on an annual basis," Dr. Brian Currie said Tuesday. Currie is vice president and medical director at Montefiore Medical Center in Bronx, New York."

Is That A Camera In Your Eye

Talk about playing Eye Spy. Wired has the story of a man who intends to place a miniature camera in his eyesocket. Don't worry, he isn't going to remove a live eye to do this. Apparently he had one removed a number of years ago. You can even see a short video of the surgery he had to extract it.

Click here for the full story or just settle for the excerpt I am going to post.

"When you completely lose an eye it is a difficult thing to let go of," he says. "The eye has an emotional attachment. It is a window to your soul."

Spence wore an eye patch for a while, which he says looked cool. But once he started thinking about having a camera in his eye, Spence got in touch with Steve Mann, a professor at the University of Toronto. Mann is one of the experts in the world of wearable computing and cyborgs -- organisms that blend natural and artificial systems.

"There are a lot of challenges in this," says Mann, "from actually building a camera system that works, to sending and receiving images, to getting the correct shape of the camera."
Even in the age of miniaturization, getting a wireless video camera into a prosthetic eye isn't easy. The shape of the prosthetic is the biggest limitation: In Spence's case, it's 9-mm thick, 30-mm long and 28-mm high.

While that might seem like plenty of room in an age when digital cameras are squeezed into unimaginably slim and compact phones, it actually isn't. The average area available inside a prosthetic eye for an imaging sensor is only about 8 square mm, explains Phil Bowen, an ocularist who is working with Spence. Also, a digital camera has many more components than the visible lens and the sensor behind it, including the power supply and image-processing circuitry. Getting a completely self-contained camera module to fit into the tiny hollow of a prosthetic eye is a significant engineering challenge.

That's where Professors Huang and Rogers' research could come in handy. Three months ago, the duo published a paper that showed how a new sensor built out of a flexible mesh of wire-connected pixels could replace the traditional flat imaging chip as the light sensor for a camera. The mesh is made from many of the same materials as a standard digital-camera sensor, but it has the ability to conform to convoluted, irregular surfaces -- like the back of a synthetic eyeball.
"Our cameras might more naturally integrate with a prosthetic eye, due to their hemispherical shapes," says Rogers. "One might also argue that they can provide a more human-like perception of the world."

Then there's the question of how the prosthetic eyeball (the outer shell for the camera) will be made. The eyeball chassis has to close shut and be watertight.

Traditional prosthetic eyes are single pieces made with polymethyl-methacrylate (PMMA), a flexible polymer that is also used in dentures. To fit a camera in, Bowen redesigned the prosthetic eye into two pieces that could snap shut.

But with a camera inside there's something new to worry about. The modified prosthetic eye will be heavier than traditional ones and that could affect the eye socket, says Bowen. "The weight might stretch out the lower lid," he says, potentially disfiguring the face.

Medical Technology- The future is now

In my travels throughout the web I often search for interesting gadgets and inventions that will change our lives. A while back I stumbled onto MedGadget. It is an interesting site that discusses technological advances in medicine.

Here are a couple of things that caught my eye:

MIT Technology Review is reporting on new metal and polymer microgrippers that can be chemically activated to grab or cut tissue deep within the body without requiring any incisions. The scientists that developed the device envision swallowing a bunch of these and then guiding the particles using magnets to specific spots in the body for microsurgeries or doing biopsies.

From MIT Tech Review:

A gripper based on the current design could respond autonomously to chemical cues in the body. For example, it might react to the biochemicals released by infected tissue by closing around the tissue, so that pieces can be removed for analysis.
Gracias [David Gracias, biomolecular and chemical-engineering professor at JHU] and his colleagues presented the microgripper at the American Chemical Society meeting earlier this month. To demonstrate the device, they used it to grasp and maneuver tiny beads and clumps of cells in a petri dish. They have also used the device in the laboratory to perform an in vitro biopsy on a cow's bladder. "This is the first micromachine that has been shown convincingly to do very useful things," Gracias says. "And it does not require electric power for operation."

The open gripper is 500 micrometers (0.05 centimeters) in diameter, and it is made of a film of copper and chromium covered with polymer. As long as the polymer stays rigid, the gripper remains open. But introducing a chemical trigger or lowering the temperature causes the polymer to soften, actuating the gripper's fingers so that they curl inward to form a ball that is 190 micrometers wide. Another chemical signal can be used to reopen the gripper. All of the chemicals used as triggers in experiments are harmless to the body.

And the ReWalk Exoskeleton which helps paralyzed people walk again. It is made by an Israeli company called Argo Medical Technologies. I think this is just very cool.

ReWalk™, the first commercially viable upright walking assistance tool, enables wheelchair users with lower-limb disabilities to stand, walk, and even climb stairs. For potentially millions of wheelchair users.

There is a cool video you can see by clicking on this link.

A Whale of an Invention

I thought that that this is very cool:

LONDON, United Kingdom (CNN) -- Medicine has much to learn from nature. There are literally millions of medical compounds out there that could cure diseases, help improve treatment and even protect us from some types of bacteria.cientists have been tapping into nature's resources for inspiration on how to treat humans.

Humpback whales, sea cucumbers and Australian red algae are just a few of the species leading the way in modern medicine.

The humpback whale has a design within its heart that could help save the lives of many patients suffering from heart disease.

With a heart that can pump six bath tubs of blood around a circulation system that is 4,500 times as complex as our own, and in only three heartbeats a minute, it has fascinated scientists as to how it manages this feat.

But it was while studying how the whale's heart is able to do this that Dr Jorge Reynolds-- (who placed the first external pacemaker in the body of a priest who survived for an additional 17 years) discovered nano-sized 'wires'. These wires allow electrical signals to stimulate the heartbeats even through masses of non-conductive blubber.

This discovery could be the key to replacing the traditional pacemaker, scientists say. Instead of having to install a battery-powered pacemaker the whale 'wires' could be used to stimulate heart beats.

Whale 'wires' could save the extra bouts of surgery, which are currently needed to replace the batteries in pacemakers.

It doesn't end there. There's also the added bonus of saving money. With the worldwide market for pacemakers expected to reach $3.7 billion by 2010, this technology, which costs only a few cents to make, could replace pacemakers and save billions.

At Ohio's Cleveland West Reserve University Jeffrey Capadona has pioneered the creation of a material that could help treat Parkinson's disease, stroke and spinal chord injuries.

This time the inspiration was the humble sea cucumber, whose skin can change from a rigid to flexible state with ease.

Capadona argues that tiny electrodes implanted into the brain are sometimes used to treat Parkinson's disease, stroke and spinal chord injuries. But they can become less effective over time as the body creates scar tissue around the hard implant.

Using this new material, which was based on the skin of sea cucumber, could improve treatment as the material can become less rigid and prolong its effectiveness.

Even red algae in Australia have provided inspiration to scientists who now believe they could help control some diseases.

Researchers from the University of New South Wales, Australia, discovered that the red algae found just off the coast was free from biofilms-- a congregation of bacteria that are the cause of 70 percent of all human infections.

Read the whole thing.

Sarcasm Seen as Evolutionary Survival Skill

Oh really. I never would have guessed.

"Humans are fundamentally social animals. Our social nature means that we interact with each other in positive, friendly ways, and it also means we know how to manipulate others in a very negative way.

Neurophysiologist Katherine Rankin at the University of California, San Francisco, has also recently discovered that sarcasm, which is both positively funny and negatively nasty, plays an important part in human social interaction.

So what?

I mean really, who cares? Oh for God's sake. Don’t you have anything better to do that read this column?

According to Dr. Rankin, if you didn’t get the sarcastic tone of the previous sentences you must have some damage to your parahippocampal gyrus which is located in the right brain. People with dementia, or head injuries in that area, often lose the ability to pick up on sarcasm, and so they don’t respond in a socially appropriate ways.

Presumably, this is a pathology, which in turn suggests that sarcasm is part of human nature and probably an evolutionarily good thing.

How might something so, well, sarcastic as sarcasm, be part of the human social toolbox?"

For the full story please click here.

Lack of Sleep is Dangerous

Can't say that I am really all that surprised by any of this. HealthDay reports on the importance of sleep. The article discusses compares modern sleep habits to the past and how attempting to improve productivity by cutting down on sleep is problematic.

"For healthy people, there's a big temptation to voluntarily restrict sleep, to stay up an hour or two or get up an hour or two earlier," said Dr. Greg Belenky, director of the Sleep and Performance Research Center at Washington State University Spokane.

"But you're really reducing your productivity and exposing yourself to risk," Belenky added.

That's a message doctors are trying to spread to Americans, including the estimated 40 million people who struggle with some type of sleep disorder each year.

Before Thomas Edison invented the light bulb in 1880, people slept an average of 10 hours a night. These days, Americans average 6.9 hours of sleep on weeknights and 7.5 hours a night on weekends, according to the National Sleep Foundation.

"The group of people getting optimal sleep is getting smaller and smaller," said Dr. Chris Drake, senior scientist at the Henry Ford Hospital Sleep Disorders and Research Center in Detroit. "When a person's sleep drops to six hours or less, that's when a lot of things become very problematic."

While experts recommend seven to eight hours of sleep each night, the amount needed for an individual can vary.

But lack of sleep affects a person in one of two ways, Belenky said. First, sleeplessness influences the day-to-day performance of tasks.

"The performance effects are seen immediately," he said. "You short-change yourself of sleep, and you see the effects immediately. You can make a bad decision. You can miss something. Have a moment's inattention, and you're off the road."

The longer-term effects of sleep deprivation involve a person's health. Doctors have linked lack of sleep to weight gain, diabetes, high blood pressure, heart problems, depression and substance abuse.

"Hormones that process appetite begin to get disorganized," said Drake, who's also an assistant professor of psychiatry and behavioral neuroscience at the Wayne State University School of Medicine. There's a decrease in the amount of leptin, an appetite-suppressing hormone, when a person gets too little sleep. At the same time, ghrelin -- a hormone that stimulates appetite -- increases with a lack of sleep.

Too little sleep also interferes with the body's ability to regulate glucose and can cause inflammation leading to heart problems and a rise in blood pressure. "There's a stress response to being in a sleep loss," Belenky said.

The types of people not getting enough sleep also break down into two groups. First, there are those who make the conscious choice to go without enough sleep.

"It's sort of part of the culture," Belenky said. "People pride themselves on getting little sleep. You'll hear people bragging, 'I only need six hours a night.' So there's a macho element here."

On the other hand, there are people who are suffering from sleep disorders. These disorders include:

• Insomnia, an inability to go to sleep or stay asleep.
• Sleep apnea, or breathing interruptions during sleep that cause people to wake up repeatedly.
• Restless legs syndrome, a tingling or prickly sensation in the legs that causes a person to need to move them, interrupting sleep."

For the full story please click here.

Wanted: Women to eat chocolate For a Year

Who wants to volunteer for this.

LONDON, England (CNN) -- Scientists in the UK are seeking 150 women to eat chocolate every day for a year in the cause of medical research.

The trial, at the University of East Anglia in Norwich, eastern England, will test whether a natural compound found in cocoa, the main ingredient of chocolate, could cut the risk of heart disease among women with diabetes.

A Belgian confectionist has created the special chocolate bar containing high levels of flavonoids -- a plant compound that has been shown to reduce heart risk factors -- to be used in the experiment. Soy, another natural source of flavonoids, has also been added to the bar.

Participants, who must be postmenopausal women under the age of 70, will have their risk of heart disease tested on five occasions during the year to see whether change occurs.

"The hypothesis of this exciting study is that flavonoids may improve the level of protection against heart disease over and above that provided by conventional drugs," said Dr. Ketan Dhatariya, a consultant in diabetes at the Norfolk and Norwich University Hospital.

"If the trial confirms this, it could have a far-reaching impact on the advice we give to postmenopausal women who have type 2 diabetes."

For the full story click here.

A Guide to Predicting Your Medical Future

Newsweek has an interesting article called A Guide to Predicting Your Medical Future. In personal health news the wife thinks I am crazy to consider trying out for American Gladiators.

Every man needs to have a mountain to climb. This just might be one of mine.

I Really Should Sleep More

Reuters reports that lack of sleep can be deadly.

People who do not get enough sleep are more than twice as likely to die of heart disease, according to a large British study released on Monday.

Although the reasons are unclear, researchers said lack of sleep appeared to be linked to increased blood pressure, which is known to raise the risk of heart attacks and stroke.

A 17-year analysis of 10,000 government workers showed those who cut their sleeping from seven hours a night to five or less faced a 1.7-fold increased risk in mortality from all causes and more than double the risk of cardiovascular death.

Study Links Diet Soft Drinks With Cardiac Risk

MONDAY, July 23 (HealthDay News) -- Drinking more than one soda a day -- even if it's the sugar-free diet kind -- is associated with an increased incidence of metabolic syndrome, a cluster of risk factors linked to the development of diabetes and cardiovascular disease, a study finds.

The link to diet soda found in the study was "striking" but not entirely a surprise, said Dr. Ramachandran Vasan, study senior author and professor of medicine at Boston University School of Medicine. There had been some hints of it in earlier studies, he said.

"But this is the first study to show the association in a prospective fashion and in a large population," Vasan said.

That population consisted of more than 6,000 participants in the Framingham Heart Study, which has been following residents of a Massachusetts town since 1948. When the soda portion of the study began, all participants were free of metabolic syndrome, a collection of risk factors including high blood pressure, elevated levels of the blood fats called triglycerides, low levels of the artery-protecting HDL cholesterol, high fasting blood sugar levels and excessive waist circumference. Metabolic syndrome is the presence of three or more of these risk factors.

Over the four years of the study, people who consumed more than one soft drink of any kind a day were 44 percent more likely to develop metabolic syndrome than those who didn't drink a soda a day.

That sound you hear is the collective groaning of Diet Coke addicts all over the world.

He Is Not Really Dead

The latest issue of Newsweek has an interesting article called Back From the Dead. Some of you may be aware that I spent seven years working as a CPR and First Aid Instructor so these sorts of articles are of interest to me.

This is a story about what happens when your heart stops: about new research into how brain cells die and how something as simple as lowering body temperature may keep them alive—research that could ultimately save as many as 100,000 lives a year. And it's about the mind as well, the visions people report from their deathbeds and the age-old questions about what, if anything, outlives the body. It begins with a challenge to something doctors have always been taught in medical school: that after about five minutes without a pulse, the brain starts dying, followed by heart muscle—the two most voracious consumers of oxygen in the body, victims of their own appetites. The emerging view is that oxygen deprivation is merely the start of a cascade of reactions within and outside the cells that can play out over the succeeding hours, or even days. Dying turns out to be almost as complicated a process as living, and somehow, among its labyrinthine pathways

I find this to be fascinating. Not unlike so many others I have wondered what happens when you die. Where does your mind go? Do you feel any pain? Do you have any understanding of what is happening? Do you go off into the next whatever with the words, sounds and noises of those that were around you?

Becker's interest in mitochondria reflects a new understanding about how cells die from loss of circulation, or ischemia. Five minutes without oxygen is indeed fatal to brain cells, but the actual dying may take hours, or even days. Doctors have known for a long time that the consequences of ischemia play out over time. "Half the time in cardiac arrest, we get the heart going again, blood pressure is good, everything is going along," says Dr. Terry Vanden Hoek, director of the Emergency Resuscitation Center at the University of Chicago, "and within a few hours everything crashes and the patient is dead." It took some time, though, for basic research to supply an explanation. Neumar, working with rats, simulates cardiac arrest and resuscitation, and then examines the neurons at intervals afterward. Up to 24 hours later they appear normal, but then in the next 24 hours, something kicks in and they begin to deteriorate. And Dr. James R. Brorson of the University of Chicago has seen something similar in neural cells grown in culture; deprive them of oxygen and watch for five minutes, or even much longer, and not much happens. "If your car runs out of gas, your engine isn't destroyed, it just needs fuel," he says.

Cell death isn't an event; it's a process. And in principle, a process can be interrupted. The process appears to begin in the mitochondria, which control the cell's self-destruct mechanism, known as apoptosis, and a related process, necrosis. Apoptosis is a natural function, destroying cells that are no longer needed or have been damaged in some way. Cancer cells, which might otherwise be killed by apoptosis, survive by shutting down their mitochondria; cancer researchers are looking for ways to turn them back on. Becker is trying to do the opposite, preventing cells that have been injured by lack of oxygen from, in effect, committing suicide.

It's a daunting problem. "We're asking the questions," says one leading researcher, Dr. Norm Abramson of the University of Pittsburgh. "We just haven't found the answers." Until recently, the conventional wisdom was that apoptosis couldn't be stopped once it was underway. It proceeds by a complex sequence of reactions—including inflammation, oxidation and cell-membrane breakdown—none of which seems to respond to traditional therapies. Becker views cell death in cardiac arrest as a two-step process, beginning with oxygen deprivation, which sets up the cell for apoptosis; then the heart starts up again and the patient gets a lungful of oxygen, triggering what is called reperfusion injury. The very substance required to save the patient's life ends up injuring or killing him.

I truly do not fear death. How can I fear it? I don't really know anything about it. Don't misunderstand, I don't want to die. When I said that I want to live for a thousand years it is because I have so many interests. There is so much to do and so very little time.

To quote my grandfather I'll fight for every last breath because I can and because I am. It doesn't have to make sense to you, but it does to me.

My children are a huge part of my interest in living. It is not just because I can't imagine not being there for them but because I am intensely curious about who they are going to become. When they grow up who will they be. What will they do and with whom?

Anyway, I think that the article is quite interesting. Give it a read.

Walgreens- Unfair Pricing

Over at the Freakonomics blog they have a post that is quite disturbing.

Many of his patients, he explained, must pay for their drugs out-of-pocket, and yet even the generic drugs at pharmacy chains like Walgreens, Eckerd, and CVS could cost them dearly.

So Wolf began snooping around and found that two chains, Costco and Sam’s Club, sold generics at prices far, far below the other chains. Even once you factor in the cost of buying a membership at Costco and Sam’s Club, the price differences were astounding. Here are the prices he found at Houston stores for 90 tablets of generic Prozac:

Walgreens: $117

Eckerd: $115

CVS: $115

Sam’s Club: $15

Costco: $12

Those aren’t typos. Walgreens charges $117 for a bottle of the same pills for which Costco charges $12.

I was skeptical at first. Why on earth, I asked Wolf, would anyone in his right mind fill his generic prescription at Walgreen’s instead of Costco?

His answer: if a retiree is used to filling his prescriptions at Walgreens, that’s where he fills his prescriptions — and he assumes that the price of a generic drug (or, perhaps, any drug) is pretty much the same at any pharmacy. Talk about information asymmetry; talk about price discrimination.

Read the whole story.

Chest presses, not breaths, help CPR

In a different life I spent a number of years teaching CPR and First AID .

Chest compression — not mouth-to-mouth resuscitation — seems to be the key in helping someone recover from cardiac arrest, according to new research that further bolsters advice from heart experts.

A study in Japan showed that people were more likely to recover without brain damage if rescuers focused on chest compressions rather than rescue breaths, and some experts advised dropping the mouth-to-mouth part of CPR altogether. The study was published in Friday's issue of the medical journal The Lancet.

More than a year ago, the
American Heart Association revised CPR guidelines to put more emphasis on chest presses, urging 30 instead of 15 for every two breaths given. Stopping chest compressions to blow air into the lungs of someone who is unresponsive detracts from the more important task of keeping blood moving to provide oxygen and nourishment to the brain and heart.

Another big advantage to dropping the rescue breaths: It could make bystanders more willing to provide CPR in the first place. Many are unwilling to do the mouth-to-mouth part and become flummoxed and fearful of getting the ratio right in an emergency.

Sudden cardiac arrest — when the heart suddenly stops beating — can occur after a heart attack or as a result of electrocution or near-drowning. It's most often caused by an abnormal heart rhythm. The person experiencing it collapses, is unresponsive to gentle shaking and stops normal breathing.

In the new study, researchers led by Dr Ken Nagao of Surugadai Nihon University Hospital in Tokyo analyzed 4,068 adult patients who had cardiac arrest witnessed by bystanders. Of those, 439 received chest compressions only from bystanders, and 712 received conventional CPR — compressions and breaths.

Any CPR attempt improved survival odds. However, 22 percent of those who received just chest compressions survived with good neurological function compared with only 10 percent of those who received combination CPR.

Click here to read the whole story.

Regrow Lost Limbs

This article is pretty cool.

NEW YORK - Researchers are trying to find ways to regrow fingers — and someday, even limbs — with tricks that sound like magic spells from a
Harry Potter novel.There's the guy who sliced off a fingertip but grew it back, after he treated the wound with an extract of pig bladder. And the scientists who grow extra arms on salamanders. And the laboratory mice with the eerie ability to heal themselves.

This summer, scientists are planning to see whether the powdered pig extract can help injured soldiers regrow parts of their fingers. And a large federally funded project is trying to unlock the secrets of how some animals regrow body parts so well, with hopes of applying the the lessons to humans.

Take a look at this:

Up to about age 2, people can consistently regrow fingertips, says Dr. Stephen Badylak, a regeneration expert at the University of Pittsburgh. But that's rare in adults, he said.

Spievack, however, did have a major advantage — a brother, Alan, a former Harvard surgeon who'd founded a company called ACell Inc., that makes an extract of pig bladder for promoting healing and tissue regeneration.

It helps horses regrow ligaments, for example, and the federal government has given clearance to market it for use in people. Similar formulations have been used in many people to do things like treat ulcers and other wounds and help make cartilage.

The summer before Lee Spievack's accident, Dr. Alan Spievack had used it on a neighbor who'd cut his fingertip off on a tablesaw. The man's fingertip grew back over four to six weeks, Alan Spievack said.

Lee Spievack took his brother's advice to forget about a skin graft and try the pig powder.

Soon a shipment of the stuff arrived and Lee Spievack started applying it every two days. Within four weeks his finger had regained its original length, he says, and in four months "it looked like my normal finger."

Spievack said it's a little hard, as if calloused, and there's a slight scar on the end. The nail continues to grow at twice the speed of his other nails.

"All my fingers in this cold weather have cracked except that one," he said.

All in all, he said, "I'm quite impressed."

For the full story please click here.